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It has been clear that due to the lack of epigenetic, transcriptional, and translational regulation of eryptosis, ion transport and adjustment of cation channels to fine-tune ion fluxes have come to the forefront in this RCD of erythrocytes

Research has shown that Bax by itself is capable of inducing pore formation in the mitochondrial membrane, following interaction with cardiolipin ( Table 1 provides a list of proteins that can form oligomers and interact with cardiolipin to induce pore formation and disease processes, with associated references

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